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Sickle Cell Anemia Conclusion

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... asked about drug use and whether they have sickle-cell anemia (Barker, 2001). Michigan. While a pediatric resident, Dr. Perricone researched sickle cell anemia using glycolic acid as a therapeutic Women at risk for transmitting genetic disorders (eg, sickle cell disease, Tay-Sachs disease, cystic fibrosis) should be genetic vulnerabilities to certain illnesses (such as, for instance, sickle cell anemia) as well as possible treatments basis of ethnicity include Tay-Sachs and Canavan diseases, thalassemias, and sickle cell anemia. The most common ...



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Sources list for SICKLE CELL ANEMIA CONCLUSION:

Charache, et. al. 1995. Effect of hydroxyurea on the frequency of painful crises in sickle cell anemia. Investigators of the Multicenter Study of Hydroxyurea in Sickle Cell Anemia. N Engl J Med 332:1317-22.
Sickle Cell Disease

Seltzer, WK; Abshire, TC; Lane, PA; Roloff, JS; Githens, JH. 1992. Molecular genetic studies in black families with sickle cell anemia and unusually high levels of fetal hemoglobin. Hemoglobin 16: 363-77.
Sickle Cell Disease

Powars, D, et. al. 1994. Beta-S gene cluster haplotypes modulate hematologic and hemorheologic expression in sickle cell anemia. Use in predicting clinical severity. Am. J. Ped. Hematology-Oncology 16:55-61.
Sickle Cell Disease

Powars, D, et al. 1993. Sickle cell anemia. Beta s gene cluster haplotypes as genetic markers for severe disease expression. Am. J. Dis. Child. 147:1197-1202.
Sickle Cell Disease

Spires R. Ocular manifestations of sickle cell disease. J Ophthalmic Nurs Technol 1995;14:74-7.
Sickle Cell Retinopathy

 


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